You Searched For: Fmoc-2-aminobutyric+acid


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Supplier: Apollo Scientific
Description: 3-(Fmoc-amino)-3-(1-Boc-3-piperidyl)propanoic acid

Catalog Number: (APOSOR470325-5G)
Supplier: Apollo Scientific
Description: 3-(Fmoc-amino)-3-(3-tert-butoxyphenyl)propanoic acid
UOM: 1 * 5 g


Catalog Number: (APOSBIPG1403-100MG)
Supplier: Apollo Scientific
Description: Fmoc-N-amido-PEG9-acid
UOM: 1 * 100 mg


Supplier: Thermo Fisher Scientific
Description: (R)-3-(Fmoc-amino)-3-(2-nitrophenyl)propionic acid 98%
Supplier: Apollo Scientific
Description: 2-(Fmoc-amino)-2-(4-tetrahydrothiopyranyl)acetic acid

Catalog Number: (BOSSBS-1302R-CY5)
Supplier: Bioss
Description: Glutamic Acid Decarboxylase (GAD) catalyzes the conversion of L glutamate to g-aminobutyric acid (GABA), the principal inhibitory neurotransmitter in the brain, and a putative paracrine signal molecule in pancreatic islets. GAD has a restricted tissue distribution. It is highly expressed in the cytoplasm of GABAergic neurons in the central nervous system (CNS) and pancreatic beta cells. It is also present in other non-neuronal tissues such as testis, oviduct and ovary. GAD is also transiently expressed in non-GABAergic cells of the embryonic and adult nervous system, suggesting its involvement in development and plasticity. GAD exists as two isoforms, GAD65 and GAD67 (molecular masses of 65 and 67 kD, respectively) that are encoded by two different genes. GAD65 is an ampiphilic, membraneanchored protein, (585 amino acid residues) and is encoded on human chromosome 10. GAD67 is a cytoplasmic protein (594 amino acid residues) and is encoded on chromosome 2. There is 64% amino acid identity between the two isoforms, with the highest diversity located at the N terminus, which in GAD65 is required for targeting the enzyme to GABA-containing secretory vesicles. The two isoforms appear to have distinct intraneuronal distribution in the brain. GAD65 has been identified as an autoantigen in insulindependent diabetes mellitus (IDDM) and stiff-man syndrome (SMS), IDDM is an autoimmune disease that results from T cell mediated destruction of pancreatic insulin-secreting beta cells. Islet-reactive T cells and primarily to GAD65 (also named beta cell autoantigen) can be detected in peripheral blood of 80% of recent-onset IDD patients and in pre-diabetic high-risk subjects before onset of clinical symptoms. This suggests that GAD may be an important marker in the early stages of the disease.
UOM: 1 * 100 µl


Catalog Number: (BOSSBS-1302R-CY7)
Supplier: Bioss
Description: Glutamic Acid Decarboxylase (GAD) catalyzes the conversion of L glutamate to g-aminobutyric acid (GABA), the principal inhibitory neurotransmitter in the brain, and a putative paracrine signal molecule in pancreatic islets. GAD has a restricted tissue distribution. It is highly expressed in the cytoplasm of GABAergic neurons in the central nervous system (CNS) and pancreatic beta cells. It is also present in other non-neuronal tissues such as testis, oviduct and ovary. GAD is also transiently expressed in non-GABAergic cells of the embryonic and adult nervous system, suggesting its involvement in development and plasticity. GAD exists as two isoforms, GAD65 and GAD67 (molecular masses of 65 and 67 kD, respectively) that are encoded by two different genes. GAD65 is an ampiphilic, membraneanchored protein, (585 amino acid residues) and is encoded on human chromosome 10. GAD67 is a cytoplasmic protein (594 amino acid residues) and is encoded on chromosome 2. There is 64% amino acid identity between the two isoforms, with the highest diversity located at the N terminus, which in GAD65 is required for targeting the enzyme to GABA-containing secretory vesicles. The two isoforms appear to have distinct intraneuronal distribution in the brain. GAD65 has been identified as an autoantigen in insulindependent diabetes mellitus (IDDM) and stiff-man syndrome (SMS), IDDM is an autoimmune disease that results from T cell mediated destruction of pancreatic insulin-secreting beta cells. Islet-reactive T cells and primarily to GAD65 (also named beta cell autoantigen) can be detected in peripheral blood of 80% of recent-onset IDD patients and in pre-diabetic high-risk subjects before onset of clinical symptoms. This suggests that GAD may be an important marker in the early stages of the disease.
UOM: 1 * 100 µl


Catalog Number: (APOSOR307664-250MG)
Supplier: Apollo Scientific
Description: (1R,2S)-Fmoc-2-aminocyclohex-3-enecarboxylic acid
UOM: 1 * 250 mg


Supplier: Apollo Scientific
Description: Fmoc-2-amino-4,4,4-trifluorobutyric acid

Supplier: Apollo Scientific
Description: Fmoc-trans-4-aminocyclohexane-1-carboxylic acid 99%

Supplier: Apollo Scientific
Description: Fmoc-N-amido-dPEG3-acid

Catalog Number: (PRSI28-272)
Supplier: ProSci Inc.
Description: The gamma-aminobutyric acid (GABA) A receptor is a multisubunit chloride channel that mediates the fastest inhibitory synaptic transmission in the central nervous system. This gene encodes GABA A receptor, beta 2 subunit. It is mapped to chromosome 5q34 in a cluster comprised of genes encoding alpha 1 and gamma 2 subunits of the GABA A receptor.
UOM: 1 * 100 µG


Catalog Number: (APOSOR312038-1G)
Supplier: Apollo Scientific
Description: 3-Aminooxetane-3-carboxylic acid, N-FMOC protected 97%
UOM: 1 * 1 g


Catalog Number: (APOSOR2679-250MG)
Supplier: Apollo Scientific
Description: Fmoc-Cyclopropylglycine
UOM: 1 * 250 mg


Supplier: Apollo Scientific
Description: cis-3-Aminocyclohexanecarboxylic acid, N-FMOC protected

Supplier: Apollo Scientific
Description: Fmoc-3-(aminooxy)acetic acid ≥98%

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Stock for this item is limited, but may be available in a warehouse close to you. Please make sure that you are logged in to the site so that available stock can be displayed. If the call is still displayed and you need assistance, please call us on +353 1 88 22222.
This product is marked as restricted and can only be purchased by approved Shipping Accounts. If you need further assistance, email VWR Regulatory Department at eurega_services@eu.vwr.com
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